Search results for "Central Nervous System Depressants"

showing 10 items of 33 documents

Morphologic variations in the pineal gland of the albino rat after a chronic alcoholisation process.

2018

We studied the effect of alcohol on the pineal gland of 48 male Wistar rats. Animals were divided into control and experimental groups. The experimental group underwent a previous progressive alcoholisation period with ethanol diluted in water at a concentration of 40%. Animals were sacrificed at 3, 6, 9 and 12 months, and the ultrastructure, karyometric indices, and number of synaptic bodies in the pineal gland were analysed. The results showed progressive morphologic alterations in the ethanol-treated animals, which culminated in fatty degeneration of the pineal parenchyma after 6 months. The karyometric indices decreased in both the central and peripheral areas compared with the control …

0301 basic medicineMalemedicine.medical_specialtyPeriod (gene)BiologyPineal ParenchymaPineal Gland03 medical and health sciencesPineal gland0302 clinical medicineMicroscopy Electron TransmissionInternal medicinemedicineAnimalsCircadian rhythmRats WistarEthanolCentral Nervous System DepressantsCell BiologyGeneral MedicineChronic alcoholCircadian RhythmRats030104 developmental biologymedicine.anatomical_structureEndocrinologyUltrastructure030217 neurology & neurosurgeryDevelopmental BiologyTissuecell
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Acute Alcohol Effects on Neuronal and Attentional Processing: Striatal Reward System and Inhibitory Sensory Interactions under Acute Ethanol Challenge

2004

The acute influence of ethanol on cerebral activity induces complex psycho-physiological effects that are considerably more pronounced during acute ethanol influx than during maximal blood alcohol concentration (elimination phase). Despite the psychiatric and forensic relevance of these different ethanol effects, the underlying neuronal mechanisms are still unclear. In total, 20 male healthy volunteers were investigated each with three different experimental conditions in a randomized order using an intravenous ethanol challenge (40 g bolus infusion): during influx phase, elimination phase, and under placebo condition. During and after the ethanol (or placebo) infusion, neuropsychological t…

AdultMaleCentral nervous systemSensory systemStriatumNeuropsychological TestsPlaceboRewardFluorodeoxyglucose F18Cortex (anatomy)Image Processing Computer-AssistedmedicineHumansAttentionSingle-Blind MethodSensory cortexBrain ChemistryNeuronsPharmacologyTemporal cortexEthanolCentral Nervous System DepressantsReciprocal inhibitionNeostriatumPsychiatry and Mental healthGlucosemedicine.anatomical_structurePsychologyNeuroscienceTomography Emission-ComputedNeuropsychopharmacology
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A fatal paramethoxymethamphetamine intoxication.

2003

During the last years in Germany a marked increase in the use of amphetamines such as 3,4-methylenedioxymethamphetamine (MDMA, ecstasy) has been observed. The use of these recreational drugs is especially common among young people participating in rave parties. Occasionally ring-methoxylated phenethylamine derivatives like paramethoxymethamphetamine (PMMA) or paramethoxyamphetamine (PMA) are found in street drugs offered as ecstasy. These compounds exhibit a higher toxicity than the methylenedioxyamphetamine derivatives. We report on the death of a 22-year-old man after the ingestion of ecstasy pills containing PMMA and PMA. The PMMA concentration in femoral blood was 0.85 mg/l. Besides PMA…

AdultMalemedicine.drug_classEcstasyPharmacologyGas Chromatography-Mass SpectrometryPathology and Forensic MedicineMethamphetaminechemistry.chemical_compoundCocaineDopamine Uptake InhibitorsmedicineIngestionHumansAmphetamineChromatography High Pressure LiquidEthanolbusiness.industryIllicit DrugsAmphetaminesForensic toxicologyCentral Nervous System DepressantsMDMADesigner drugIssues ethics and legal aspectsAmphetaminechemistryPillBenzoylecgonineCentral Nervous System Stimulantsbusinessmedicine.drugLegal medicine (Tokyo, Japan)
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Levels of GHB in hair after regular application.

2021

Gamma hydroxybutyrate (GHB) is a central nervous system depressant that is an approved drug for the treatment of narcolepsy with cataplexy and other syndromes. Due to its dose dependent stimulating, relaxing or sedative effects, illicit abuses include recreational use by young people and cases of drug-facilitated crime (DFC). Since GHB is also produced endogenously, for forensic questions, it is important to be able to differentiate between endogenous GHB and elevated levels due to additional intake. In this study, we measured GHB concentrations in hair of patients with narcolepsy receiving daily GHB treatment. The results were compared to endogenous concentrations and concentrations after …

AdultMalemedicine.medical_specialtyAdolescentSodium OxybateDose dependenceRecreational use01 natural sciencesPathology and Forensic Medicine03 medical and health sciencesYoung Adult0302 clinical medicineTandem Mass SpectrometryInternal medicineMedicineHumans030216 legal & forensic medicineNarcolepsySleep disorderbusiness.industry010401 analytical chemistryCentral Nervous System DepressantsGamma hydroxybutyratemedicine.disease0104 chemical sciencesSodium saltEndocrinologySedative EffectsFemalebusinessSodium OxybateLawNarcolepsyChromatography LiquidHairForensic science international
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Apomorphine-Induced Growth Hormone Response Is Attenuated by Ethanol but Not Dextromethorphan

2007

Background: Misuse of alcohol drinking is a major health problem. Alcohol decreases spontaneous growth hormone (GH) secretion, but the mechanism is unclear. The aim of this study was to test whether administration of alcohol (study 1) or a N-methyl d-aspartate (NMDA) receptor antagonist (study 2) attenuates the GH response to pharmacological dopaminergic stimulation. Methods: The 2-session repeated measures design was conducted at the endocrine laboratory at the Department of Psychiatry at the Free University Berlin. Twenty healthy Caucasian males aged 35±10 years without a history of alcohol use disorders were tested using the Apomorphine (APO) challenge test. In study 1, we injected APO (…

AdultMalemedicine.medical_specialtyApomorphineMedicine (miscellaneous)PharmacologyToxicologyPlaceboDextromethorphanReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundOral administrationInternal medicinemedicineHumansEthanolEthanolHuman Growth Hormonebusiness.industryDopaminergicCentral Nervous System DepressantsDextromethorphanGrowth hormone secretionApomorphinePsychiatry and Mental healthEndocrinologychemistryArea Under CurveDopamine AgonistsNMDA receptorbusinessmedicine.drugAlcoholism: Clinical and Experimental Research
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Systemic administration of D-penicillamine prevents the locomotor activation after intra-VTA ethanol administration in rats.

2010

Although recently published studies seem to confirm the important role displayed by acetaldehyde (ACH), the main metabolite of ethanol, in the behavioral effects of ethanol, the origin of ACH is still a matter of debate. While some authors confer more importance to the central (brain metabolism) origin of ACH, others indicate that the hepatic origin could be more relevant. In this study we have addressed this topic using an experimental approach that combines local microinjections of ethanol into the ventral tegmental area (VTA) (which guarantees the brain origin of the ACH) to induce motor activation in rats together with systemic administration (i.p.) of several doses (0, 12.5, 25 and 50 …

AgonistLocomotor activityMalemedicine.drug_classMetaboliteCentral nervous systemAcetaldehydePharmacologyMotor Activitychemistry.chemical_compoundAlcohol-Induced Disorders Nervous SystemmedicineAnimalsRats WistarReceptorEthanolGeneral NeurosciencePenicillamineD-PenicillaminePenicillamineVentral Tegmental AreaCentral Nervous System DepressantsRatsVentral tegmental areaDAMGOBrain metabolism of ethanolDisease Models Animalmedicine.anatomical_structurechemistryBiochemistrySystemic administrationVTAmedicine.drugNeuroscience letters
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Endocytosis in cultured neurons is altered by chronic alcohol exposure.

2010

Endocytosis is required for many cellular pivotal processes, including membrane recycling, nutrient uptake, and signal transduction. This complex process is particularly relevant in polarized cells, such as neurons. Previous studies have demonstrated that alcohol alters intracellular traffic, including endocytosis, in several cell types. However, information on the effect of chronic alcohol exposure on this process in neurons is scarce. As an approach, we investigated the effect of alcohol exposure on the internalization of two widely used endocytic markers, albumin and transferrin, in developing hippocampal neurons in primary culture. The effect of this treatment on the levels of several r…

Cell signalingRHOAmedia_common.quotation_subjectEndocytic cycleNerve Tissue ProteinsEndosomesToxicologyEndocytosisClathrinHippocampusAlbuminsAnimalsInternalizationCells Culturedmedia_commonNeuronsbiologyEthanolTransferrinCentral Nervous System DepressantsBiological TransportActin cytoskeletonClathrinEndocytosisCell biologyRatsbiology.proteinFemaleIntracellularBiomarkersToxicological sciences : an official journal of the Society of Toxicology
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The pharmacological and non-pharmacological treatment of attention deficit hyperactivity disorder in children and adolescents: A systematic review wi…

2017

Background Attention deficit hyperactivity disorder (ADHD) is one of the most commonly diagnosed psychiatric disorders in childhood. A wide variety of treatments have been used for the management of ADHD. We aimed to compare the efficacy and safety of pharmacological, psychological and complementary and alternative medicine interventions for the treatment of ADHD in children and adolescents. Methods and findings We performed a systematic review with network meta-analyses. Randomised controlled trials (≥ 3 weeks follow-up) were identified from published and unpublished sources through searches in PubMed and the Cochrane Library (up to April 7, 2016). Interventions of interest were pharmacolo…

Complementary TherapiesMaleTrastorns de l'atencióPoison controllcsh:MedicineMathematical and Statistical Techniques0302 clinical medicineBehavior TherapyMedicine and Health SciencesMedicine030212 general & internal medicineChildlcsh:ScienceRandomized Controlled Trials as TopicMultidisciplinaryPharmaceuticsMethylphenidate3. Good healthGuanfacineAntidepressant Drug TherapyNeurologyTolerabilityBehavioral PharmacologyResearch DesignPhysical SciencesFemaleStatistics (Mathematics)Research Articlemedicine.drugNeurological Drug Therapymedicine.medical_specialtyAdolescentClinical Research DesignNeuropsychiatric DisordersResearch and Analysis MethodsPlacebo03 medical and health sciencesDevelopmental NeuroscienceDrug TherapyInternal medicineMental Health and PsychiatryHumansAttention deficit hyperactivity disorderPsiquiatriaStatistical MethodsAdverse effectPsychiatryPharmacologyBehaviorbusiness.industryAtomoxetinelcsh:RCentral Nervous System DepressantsBiology and Life Sciencesmedicine.diseaseAttention Deficit Disorder with HyperactivityNeurodevelopmental DisordersCentral Nervous System StimulantsAdhdlcsh:QAdverse EventsbusinessMental Health TherapiesMathematics030217 neurology & neurosurgeryNeuroscienceMeta-AnalysisPLoS ONE
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Inhibition of astroglial cell proliferation by alcohols: interference with the protein kinase C-phospholipase D signaling pathway.

2000

Abstract Ethanol inhibits astroglial cell proliferation, an effect that may contribute to the development of alcoholic embryopathy in humans. In the present study, we investigated inhibitory effects of ethanol and butanol isomers (1-, 2- and t -butanol) on astroglial cell proliferation induced by the strongly mitogenic phorbol ester, 4s-phorbol-12α,13s-dibutyrate (PDB). 4s-Phorbol-12α,13s-dibutyrate (PDB) induced a 10-fold increase of [3H]thymidine incorporation in cortical astrocytes prepared from newborn rats (EC 50 : 70 nM) which was blocked by Ro 31-8220, a cell-permeable protein kinase C (PKC) inhibitor. Ethanol blocked PDB-induced astroglial proliferation in a concentration-dependent …

IndolesButanolsPhosphatidic AcidsDiglycerideschemistry.chemical_compoundDevelopmental NeurosciencePhorbol EstersPhospholipase DAnimalsEnzyme InhibitorsProtein kinase CCells CulturedPhorbol 1213-DibutyrateProtein Kinase CEthanolEthanolCell growthPhospholipase DBrainCentral Nervous System DepressantsPhosphatidic acidequipment and suppliesIn vitroRatsEnzyme ActivationchemistryBiochemistryAstrocytesCarcinogenslipids (amino acids peptides and proteins)PhosphatidylethanolSignal transductionCell DivisionDevelopmental BiologySignal TransductionInternational journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience
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TLR4 elimination prevents synaptic and myelin alterations and long-term cognitive dysfunctions in adolescent mice with intermittent ethanol treatment.

2015

The adolescent brain undergoes important dynamic and plastic cell changes, including overproduction of axons and synapses, followed by rapid pruning along with ongoing axon myelination. These developmental changes make the adolescent brain particularly vulnerable to neurotoxic and behavioral effects of alcohol. Although the mechanisms of these effects are largely unknown, we demonstrated that ethanol by activating innate immune receptors toll-like receptor 4 (TLR4), induces neuroinflammation and brain damage in adult mice. The present study aims to evaluate whether intermittent ethanol treatment in adolescence promotes TLR4-dependent pro-inflammatory processes, leading to myelin and synapti…

MAPK/ERK pathwaySynaptic dysfunctionImmunologyNitric Oxide Synthase Type IIBrain damageHMGB1Behavioral NeuroscienceMyelinMiceCognitionmedicineAnimalsTLR4AxonHMGB1 ProteinReceptorNeuroinflammationMyelin SheathMice KnockoutMitogen-Activated Protein Kinase KinasesbiologyBinge ethanol treatmentEthanolEndocrine and Autonomic SystemsNF-kappa BCentral Nervous System DepressantsMyelin alterationsAdolescenceToll-Like Receptor 4medicine.anatomical_structureCyclooxygenase 2SynapsesTLR4biology.proteinmedicine.symptomPsychologyCognition DisordersNeuroscienceCognitive behaviorAlcohol-Related DisordersMyelin ProteinsSignal TransductionBrain, behavior, and immunity
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